Aftab et al clearly find it difficult to give up hypotheses of antidepressants (and presumably ECT) correcting biological dysfunction, speculating that antidepressants may enhance synaptic neuroplasticity. They do not seem to fully appreciate that enactive accounts of psychiatry, such as that by Sanneke de Haan (see previous post), provide a philosophical and conceptual critique of naturalistic-reductive approaches to human understanding by focusing on the person as embodied and alive (see eg. another previous post). Aftab et al’s view is what Manschrek & Kleinman (1977) called semi-critical as it doesn’t follow through sufficiently on its critique of the biomedical model (see eg. my article). As I have been arguing throughout this blog, psychiatry needs to move on from its eclectic biopsychosocial view (eg. see previous post and another and yet another) and return to Engel’s original concept of the biopsychopsychosocial model, which was a critique of reductionism in psychiatry and medicine in general (see eg. another previous post). Critical/relational psychiatry is explicitly anti-reductionist and anti-positivist whereas Aftab et al are not. Their approach to psychiatry is still not sufficiently person-centred.
Friday, April 01, 2022
Taking psychiatric critique seriously
2022) critique the philosophical and conceptual basis of an article by Read & Moncrieff (2022) about why drugs and ECT are not the answer for depression. I have sympathy with their argument that binary opposition between understanding depression as medical illness and meaningful response to life events is problematic. However, having detected this flaw, Aftab et al then justify not dealing with, even dismissing, the main focus of the Read & Moncrieff article on the effectiveness and safety of antidepressants and ECT.